Still Anemic? This Could Be Why

The mitochondrial toxin that keeps iron from reaching your cells.

Anemia is one of the most common energetic dysfunctions in the world.

If you are anemic, your cells will not get enough oxygen, and if they don’t have enough oxygen, your mitochondrial respiratory chain can’t produce enough ATP.

If you are anemic — especially if your hemoglobin is low — it is probably the case that you have not made enough heme in general, which means you haven’t made enough heme for the mitochondrial respiratory chain complexes themselves. Now your mitochondria are in double trouble.

Oxalate inhibits the deloading of transferrin-bound iron to cells, preventing them from using it to make heme for respiratory chain enzymes and hemoglobin for oxygen carrying.

The primary markers of this on an iron panel should be high iron saturation or transferrin saturation with low ferritin.

This is also expected in early stage iron overload, but if you have iron overload of any degree you shouldn’t be anemic. If anything, you should tend toward being polycythemic (too many red blood cells).

So, high iron saturation, low ferritin, and normal or high hemoglobin and red blood cell count should direct you to the iron overload protocol:

High iron saturation, low ferritin, and low hemoglobin or red blood cells should direct you to the oxalate protocol:

Another reason for high iron saturation and low ferritin could be chronically elevated hydrogen sulfide.

So, this should also drive you to the sulfur protocol:

The oxalate and sulfur protocols both have the necessary labs to distinguish between them.

If your iron saturation is low this is a major sign you instead need the iron deficiency protocol:

Get those mitochondria moving!